wald chi-square Search Results


wald chi-square  (CH Instruments)
90
CH Instruments wald chi-square
Wald Chi Square, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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wald log-linear chi-square test  (CH Instruments)
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CH Instruments wald log-linear chi-square test
Wald Log Linear Chi Square Test, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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asymptotic chi-square distribution of the wald statistic  (CH Instruments)
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CH Instruments asymptotic chi-square distribution of the wald statistic
Asymptotic Chi Square Distribution Of The Wald Statistic, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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type ii wald chi-square tests  (CH Instruments)
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CH Instruments type ii wald chi-square tests
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Type Ii Wald Chi Square Tests, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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wald chi-square test of parameter equalities  (CH Instruments)
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CH Instruments wald chi-square test of parameter equalities
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Wald Chi Square Test Of Parameter Equalities, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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wald chi-square 49.5  (CH Instruments)
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CH Instruments wald chi-square 49.5
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Wald Chi Square 49.5, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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wald chi-square statistic  (SAS institute)
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SAS institute wald chi-square statistic
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Wald Chi Square Statistic, supplied by SAS institute, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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wald chi-square statistics sas proc surveyfreq  (SAS institute)
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SAS institute wald chi-square statistics sas proc surveyfreq
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Wald Chi Square Statistics Sas Proc Surveyfreq, supplied by SAS institute, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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type iii wald chi-square  (CH Instruments)
90
CH Instruments type iii wald chi-square
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Type Iii Wald Chi Square, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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wald chi-square inclusion threshold  (CH Instruments)
90
CH Instruments wald chi-square inclusion threshold
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Wald Chi Square Inclusion Threshold, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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glmz, wald chi-square test  (CH Instruments)
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CH Instruments glmz, wald chi-square test
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Glmz, Wald Chi Square Test, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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adjusted wald chi-square  (CH Instruments)
90
CH Instruments adjusted wald chi-square
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Adjusted Wald Chi Square, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II Wald Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice

Journal: Molecular Autism

Article Title: The activation of mGluR4 rescues parallel fiber synaptic transmission and LTP, motor learning and social behavior in a mouse model of Fragile X Syndrome

doi: 10.1186/s13229-023-00547-4

Figure Lengend Snippet: Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II Wald Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice

Article Snippet: P-values for fixed effects and their interactions were obtained with type II Wald Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice

Techniques: Control, Electron Microscopy